Is it really deadly? Read at http://erowid.org/plants/amanitas/amanitas.shtml
3 day timelapse of an Amanita Muscaria.
Shot outdoors in a friends backyard in Vancouver BC Canada
For all information regarding this mushroom visit http://erowid.org/plants/amanitas/amanitas.shtml
Shot by Michel Gingras using a PD150 connected to a laptop. Sadly the original tape (DVCAM) has gone missing. The is the highest quality available!)
Was the story of Santa and his hallucinogen imbibing Reindeer a product of Amanita Muscaria psychedelic mushrooms? Join me on an expedition through Oz, within the mountainous caverns of the Gnome King - to the Tesseract symbolism in the Sci Fi channel's new "Tin Man" series. Bowie pops his head in once again - as we explore Owl symbolism and the Bohemian Grove.
If you would like a longer version of this video in DVD quality and Stereo sound, please visit my website: www.labyrinthofthepsychonaut.com
Wishing everyone a psychedelic Solstice, your humble narrator: soundlessdawn
Amanita muscaria contains a number of biologically active agents, at least two of which are known to be psychoactive. Muscimol (3hydroxy-5-aminomethy-1 isoxazole, an unsaturated cyclic hydroxamic acid) is the most significant. It is the product of the decarboxylation or drying of ibotenic acid, another important compound in the biochemistry of the fly agaric. Muscarine, discovered in 1869,[58] was long thought to be the active hallucinogenic agent in A. muscaria until the mid 20th century,[59] [60] when researchers in England,[61] Japan,[62] and Switzerland[63] recognized that these effects were due mainly to ibotenic acid and muscimol.[3]
Ibotenic acid and muscimol are structurally related to each other and to two major neurotransmitters of the central nervous system: glutamic acid and GABA respectively. Ibotenic acid and muscimol act like these neurotransmitters (muscimol is a potent GABAA agonist, while ibotenic acid is an agonist of NMDA glutamate receptors and certain metabotropic glutamate receptors[64]) which are involved in the control of neuronal activity. It is these interactions which are thought to cause the psychoactive effects found in intoxication. Following ingestion some of the ibotenic acid is metabolised to muscimol which would appear to be the agent responsible for the majority of the psychoactivity.[5][65] Indeed, ibotenic acid's strong water solubility means that it could not distribute into the brain without an active process like a transporter; this, and the lack of any reports of permanent brain damage following A. muscaria ingestion (which would be the result of ibotenic acid entering the brain due to its effect as an NMDA receptor agonist) make it unlikely that ibotenic acid enters the brain following A. muscaria ingestion. However, it is worth noting that no studies have directly investigated ibotenic acid's ability to permeate the brain.
Muscazone is another compound more recently isolated from European specimens of the fly agaric. It is a product of the breakdown of ibotenic acid by ultra-violet radiation.[66] It is of minor pharmacological activity compared with the other agents.[5]
Muscarine binds with Muscarinic acetylcholine receptor and lead to the excitation of the neurons bearing these receptors. The levels in Amanita muscaria are minute when compared with other poisonous fungi,[67] such as the deadly Inocybe erubescens or small white Clitocybe species C. dealbata and C. rivulosa, and are too insignificant to play a role in the symptoms of poisoning.[68]
Amanita muscaria and related species are known as effective accumulators of vanadium (up to 500 mg/kg in dry weight). Vanadium is present as an organometallic compound (called amavadine) in fruit-bodies. However, the biological importance of the accumulation process is unknown.
Presentazione di una Amanita tra le più diffuse in ogni habitat, l'Amanita gialla, in questo filmato vengono fornite informazioni sui seguenti aspetti:
a - etimologia
b - strutture morfologiche
c - elementi morfocromatici generali
d - habitat
e - odore e sapore
f - commestibilità
Scheda realizzata dal Micologo Pietro Curti:
www.videofunghi.it
Presidente dell'Associazione AMINT:
www.amint.it
Hunting Amanita calyptrata (aka A. lanei) in Oregon's coastal mountains. WARNING: Do NOT try this at home. Amanita mushrooms can KILL YOU. Only pick edible Amanitas with a knowledgeable expert.
Presentazione del fungo letale per eccellenza, in questo filmato vengono fornite informazioni sui seguenti aspetti:
a - etimologia
b - strutture morfologiche
c - elementi morfocromatici generali
d - habitat
e - odore e sapore
f - commestibilità
Scheda realizzata dal Micologo Pietro Curti:
www.videofunghi.it
Presidente dell'Associazione AMINT:
www.amint.it
http://www.escucha.com / Amanita y los Faloides detestan las etiquetas pero les encanta actuar en directo. Puedes escuchar los MP3 y hacerte una idea, pero lo que realmente queremos es que vengas a vernos al próximo concierto http://www.amanitaylosfaloides.com
Amanita muscaria contains a number of biologically active agents, at least two of which are known to be psychoactive. Muscimol (3hydroxy-5-aminomethy-1 isoxazole, an unsaturated cyclic hydroxamic acid) is the most significant. It is the product of the decarboxylation or drying of ibotenic acid, another important compound in the biochemistry of the fly agaric. Muscarine, discovered in 1869,[58] was long thought to be the active hallucinogenic agent in A. muscaria until the mid 20th century,[59] [60] when researchers in England,[61] Japan,[62] and Switzerland[63] recognized that these effects were due mainly to ibotenic acid and muscimol.[3]
Ibotenic acid and muscimol are structurally related to each other and to two major neurotransmitters of the central nervous system: glutamic acid and GABA respectively. Ibotenic acid and muscimol act like these neurotransmitters (muscimol is a potent GABAA agonist, while ibotenic acid is an agonist of NMDA glutamate receptors and certain metabotropic glutamate receptors[64]) which are involved in the control of neuronal activity. It is these interactions which are thought to cause the psychoactive effects found in intoxication. Following ingestion some of the ibotenic acid is metabolised to muscimol which would appear to be the agent responsible for the majority of the psychoactivity.[5][65] Indeed, ibotenic acid's strong water solubility means that it could not distribute into the brain without an active process like a transporter; this, and the lack of any reports of permanent brain damage following A. muscaria ingestion (which would be the result of ibotenic acid entering the brain due to its effect as an NMDA receptor agonist) make it unlikely that ibotenic acid enters the brain following A. muscaria ingestion. However, it is worth noting that no studies have directly investigated ibotenic acid's ability to permeate the brain.
Muscazone is another compound more recently isolated from European specimens of the fly agaric. It is a product of the breakdown of ibotenic acid by ultra-violet radiation.[66] It is of minor pharmacological activity compared with the other agents.[5]
Muscarine binds with Muscarinic acetylcholine receptor and lead to the excitation of the neurons bearing these receptors. The levels in Amanita muscaria are minute when compared with other poisonous fungi,[67] such as the deadly Inocybe erubescens or small white Clitocybe species C. dealbata and C. rivulosa, and are too insignificant to play a role in the symptoms of poisoning.[68]
Amanita muscaria and related species are known as effective accumulators of vanadium (up to 500 mg/kg in dry weight). Vanadium is present as an organometallic compound (called amavadine) in fruit-bodies. However, the biological importance of the accumulation process is unknown.
Amanita muscaria contains a number of biologically active agents, at least two of which are known to be psychoactive. Muscimol (3hydroxy-5-aminomethy-1 isoxazole, an unsaturated cyclic hydroxamic acid) is the most significant. It is the product of the decarboxylation or drying of ibotenic acid, another important compound in the biochemistry of the fly agaric. Muscarine, discovered in 1869,[58] was long thought to be the active hallucinogenic agent in A. muscaria until the mid 20th century,[59] [60] when researchers in England,[61] Japan,[62] and Switzerland[63] recognized that these effects were due mainly to ibotenic acid and muscimol.[3]
Ibotenic acid and muscimol are structurally related to each other and to two major neurotransmitters of the central nervous system: glutamic acid and GABA respectively. Ibotenic acid and muscimol act like these neurotransmitters (muscimol is a potent GABAA agonist, while ibotenic acid is an agonist of NMDA glutamate receptors and certain metabotropic glutamate receptors[64]) which are involved in the control of neuronal activity. It is these interactions which are thought to cause the psychoactive effects found in intoxication. Following ingestion some of the ibotenic acid is metabolised to muscimol which would appear to be the agent responsible for the majority of the psychoactivity.[5][65] Indeed, ibotenic acid's strong water solubility means that it could not distribute into the brain without an active process like a transporter; this, and the lack of any reports of permanent brain damage following A. muscaria ingestion (which would be the result of ibotenic acid entering the brain due to its effect as an NMDA receptor agonist) make it unlikely that ibotenic acid enters the brain following A. muscaria ingestion. However, it is worth noting that no studies have directly investigated ibotenic acid's ability to permeate the brain.
Muscazone is another compound more recently isolated from European specimens of the fly agaric. It is a product of the breakdown of ibotenic acid by ultra-violet radiation.[66] It is of minor pharmacological activity compared with the other agents.[5]
Muscarine binds with Muscarinic acetylcholine receptor and lead to the excitation of the neurons bearing these receptors. The levels in Amanita muscaria are minute when compared with other poisonous fungi,[67] such as the deadly Inocybe erubescens or small white Clitocybe species C. dealbata and C. rivulosa, and are too insignificant to play a role in the symptoms of poisoning.[68]
Amanita muscaria and related species are known as effective accumulators of vanadium (up to 500 mg/kg in dry weight). Vanadium is present as an organometallic compound (called amavadine) in fruit-bodies. However, the biological importance of the accumulation process is unknown.
Amanita muscaria contains a number of biologically active agents, at least two of which are known to be psychoactive. Muscimol (3hydroxy-5-aminomethy-1 isoxazole, an unsaturated cyclic hydroxamic acid) is the most significant. It is the product of the decarboxylation or drying of ibotenic acid, another important compound in the biochemistry of the fly agaric. Muscarine, discovered in 1869,[58] was long thought to be the active hallucinogenic agent in A. muscaria until the mid 20th century,[59] [60] when researchers in England,[61] Japan,[62] and Switzerland[63] recognized that these effects were due mainly to ibotenic acid and muscimol.[3]
Ibotenic acid and muscimol are structurally related to each other and to two major neurotransmitters of the central nervous system: glutamic acid and GABA respectively. Ibotenic acid and muscimol act like these neurotransmitters (muscimol is a potent GABAA agonist, while ibotenic acid is an agonist of NMDA glutamate receptors and certain metabotropic glutamate receptors[64]) which are involved in the control of neuronal activity. It is these interactions which are thought to cause the psychoactive effects found in intoxication. Following ingestion some of the ibotenic acid is metabolised to muscimol which would appear to be the agent responsible for the majority of the psychoactivity.[5][65] Indeed, ibotenic acid's strong water solubility means that it could not distribute into the brain without an active process like a transporter; this, and the lack of any reports of permanent brain damage following A. muscaria ingestion (which would be the result of ibotenic acid entering the brain due to its effect as an NMDA receptor agonist) make it unlikely that ibotenic acid enters the brain following A. muscaria ingestion. However, it is worth noting that no studies have directly investigated ibotenic acid's ability to permeate the brain.
Muscazone is another compound more recently isolated from European specimens of the fly agaric. It is a product of the breakdown of ibotenic acid by ultra-violet radiation.[66] It is of minor pharmacological activity compared with the other agents.[5]
Muscarine binds with Muscarinic acetylcholine receptor and lead to the excitation of the neurons bearing these receptors. The levels in Amanita muscaria are minute when compared with other poisonous fungi,[67] such as the deadly Inocybe erubescens or small white Clitocybe species C. dealbata and C. rivulosa, and are too insignificant to play a role in the symptoms of poisoning.[68]
Amanita muscaria and related species are known as effective accumulators of vanadium (up to 500 mg/kg in dry weight). Vanadium is present as an organometallic compound (called amavadine) in fruit-bodies. However, the biological importance of the accumulation process is unknown.
Recorded live in concert @ The Electric Lodge, Venice, CA. 2003. Amanita is a composition by Adam Rudolph which utilizes his concept of "Cyclic Verticalism". More information @ metarecords.com and yuseflateef.com
